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Archives of Disease in Childhood Fetal and Neonatal Edition 2003;88:F455
© 2003 Archives of Disease in Childhood Fetal and Neonatal Edition

REVIEW

Jaundice in low birthweight infants: pathobiology and outcome

J F Watchko1, M J Maisels2

1 Division of Neonatology and Developmental Biology, Department of Pediatrics, University of Pittsburgh School of Medicine, Pittsburgh, Pennsylvania 15213, USA
2 Department of Pediatrics, William Beaumont Hospital, Royal Oak, MI 48073, USA

Correspondence to:
Correspondence to:
Dr Watchko
Division of Neonatology and Developmental Biology, Department of Pediatrics, Magee-Women’s Hospital, 300 Halket Street, Pittsburgh, PA 15213, USA; jwatchko{at}mail.magee.edu


ABSTRACT
Jaundice in preterm, as well as full term, infants results from (a) an increased bilirubin load in the hepatocyte, (b) decreased hepatic uptake of bilirubin from the plasma, and/or (c) defective bilirubin conjugation. Hyperbilirubinaemia in preterm infants is more prevalent, more severe, and its course more protracted than in term neonates.

Keywords: jaundice; kernicterus; prematurity

Abbreviations: TSB, total serum bilirubin; VLBW, very low birthweight




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